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［1］吴雪琼，吴长有.结核病免疫学［M］.北京：人民卫生出版社，2016.
［2］唐神结，高文.临床结核病学［M］.北京：人民卫生出版社，2011.
［3］JAMES W D，BERGER T G，ELSTON D M.安得鲁斯临床皮肤病学［M］.徐世正，译.10版.北京：科学出版社，2008.
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［17］TANDON V，MAHAPATRA A K.Management of post-tubercular hydrocephalus［J］.Childs Nerv Syst，2011，27（10）:1699-1707.
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［22］SALIE M，DAYA M，LUCAS L A，et al.Association of toll-like receptors with susceptibility to tuberculosis suggests sex-specific effects of TLR8 polymorphisms［J］.Infect Genet Evol，2015（34）:221-229.
［23］SHIN J G，PARK B L，KIM L H，et al.Association study of polymorphisms in interferon-gamma receptor genes with the risk of pulmonary tuberculosis［J］.Mol Med Rep，2015，12（1）:1568-1578.
［24］ZHANG G，WEERD N A，STIFTER S A，et al.A proline deletion in IFNAR1 impairs IFN-signaling and underlies increased resistance to tuberculosis in humans［J］.Nat Commun，2018，9（1）:85.
［25］FACCHINI L，VENTURINI E，GALLI L，et al.Vitamin D and tuberculosis:a review on a hot topic［J］.J Chemother，2015，27（3）:128-138.
［26］LEE Y H，SONG G G.Vitamin D receptor gene FokI，TaqI，BsmI，and ApaI polymorphisms and susceptibility to pulmonary tuberculosis:a meta-analysis［J］.Genet Mol Res，2015，14（3）:9118-9129.
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［28］CURTIS J，LUO Y，ZENNER H L，et al.Susceptibility to tuberculosis is associated with variants in the ASAP1 gene encoding a regulator of dendritic cell migration［J］.Nat Genet，2015，47（5）:523-527.
［29］TOBIN D M，ROCA F J，OH S F，et al.Host genotype-specific therapies can optimize the inflammatory response to mycobacterial infections［J］.Cell，2012，148（3）:434-446.